notes by dr Claudio Italiano
Here
we are talking about that strange throat pain associated with hormonal imbalance
related to thyroid hormones, because the gland is inflamed, either by bacterial
or viral infections or for reasons of an autoimmune process, with antibodies
directed against itself. Under these conditions the equilibrium of hormone
production is lost, the inflammation and the excessive blood circulation that
passes through the gland carries excess hormone with it and you can have
pictures of hyperthyroidism and then of hypothyroidism, when the gland is
transformed in a fibrous mass and no longer produces hormones; the therapy will
be treated below.
Read and understand how your doctor moves to treat you.
A separate picture deserves the thyroiditis, or the inflammatory processes that
affect the t. and can be distinguished on the basis of the etiology in bacterial
forms (pyogenic thyroiditis, tuberculous thyroiditis, and De Quervain viral, or
autoimmune (Hashimoto and Riedel's thyroiditis)
It is characterized by an acute bacterial infection, fever, throat pain,
increased volume of the gland, possible compression phenomena. The inflammatory
process leads to the release of hormone from the thyroid, so you can have
pictures of hyperthyroidism. Therapy uses antibiotics.
It recognizes a framework of viral etiology, with giant cell granulomatous
lesions of t .; due to infection with Coxachie virus, Echo, of mumps. The
clinical picture is characterized by fever, thyroid pain, inability to swallow,
increase in volume of the gland, irregular surface, lymphoplasmacellular
infiltrates of the gland, presence of giant cells. The VES rises, iodine is not
detected. Treatment is difficult, sometimes using non-steroidal
anti-inflammatory drugs.
H-thyroiditis or chronic lymphocytic or lymphomatous struma is a typical example of autoimmune thyroid disease. It is not yet known whether traumas, viral agents, irradiations and the same simple goiter are at the base of the initiation of an autoimmune process (H. thyroiditis is in fact associated with autoimmune processes such as systemic lupus, hemolytic anemias, autoimmune thrombocytopenias, anemia pernicious). The onset of the pathology consists of a slow and insidious course, with a progressive increase in the volume of the thyroid, which becomes an irregular surface, of an increased, granular consistency, with a decline in functional activity. However, inflammation sometimes results in increased circulation and accelerated hormonal uptake. The gland over time undergoes a regression phenomenon, infiltrates inflammatory cells, undergoes fibrosis and the fibrosclerotic tissue replaces the parenchymal tissue. The diagnosis makes use of the research of antimicrosomal antibodies (in 95% of cases are high) and antitireoglobulina. The therapy is based on immunosuppressive drugs but their effect is fleeting, so it is more sensible a treatment with thyroid hormones to correct the gradual deficit that the gland faces, in order to correct the rise of TSH.
It is characterized by an acute bacterial infection, fever, throat pain,
increased volume of the gland, possible compression phenomena. The inflammatory
process leads to the release of hormone from the thyroid, so you can have
pictures of hyperthyroidism. Therapy uses antibiotics.
It recognizes a framework of viral etiology, with giant cell granulomatous
lesions of t .; due to infection with Coxachie virus, Echo, of mumps. The
clinical picture is characterized by fever, thyroid pain, inability to swallow,
increase in volume of the gland, irregular surface, lymphoplasmacellular
infiltrates of the gland, presence of giant cells. The VES rises, iodine is not
detected. Treatment is difficult, sometimes using non-steroidal
anti-inflammatory drugs.
H-thyroiditis or chronic lymphocytic or lymphomatous struma is a typical example
of autoimmune thyroid disease. It is not yet known whether traumas, viral agents,
irradiations and the same simple goiter are at the base of the initiation of an
autoimmune process (H. thyroiditis is in fact associated with autoimmune
processes such as systemic lupus, hemolytic anemias, autoimmune
thrombocytopenias, anemia pernicious).
The onset of the pathology consists of a
slow and insidious course, with a progressive increase in the volume of the
thyroid, which becomes an irregular surface, of an increased, granular
consistency, with a decline in functional activity.
However, inflammation
sometimes results in increased circulation and accelerated hormonal uptake. The
gland over time undergoes a regression phenomenon, infiltrates inflammatory
cells, undergoes fibrosis and the fibrosclerotic tissue replaces the parenchymal
tissue. The diagnosis makes use of the research of antimicrosomal antibodies (in
95% of cases are high) and antitireoglobulina.
The therapy is based on immunosuppressive drugs but their effect is fleeting, so
it is more sensible a treatment with thyroid hormones to correct the gradual
deficit that the gland faces, in order to correct the rise of TSH.
It is similar to the thyroiditis of H. however it is distinguished by a
sclero-ialino process that affects the t. and that it turns into a gland of
woody consistency, low in cells and fibrous, to which compressive phenomena are
obtained on trachea, esophagus, etc.